Too much soda may raise children"s risk of fatty liver

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Source: New research provides further evidence of the harms of consuming too many sweetened beverages, after linking soda and other fructose-containing products with increased risk of liver disease.

Researchers suggest that high fructose intake, particularly from sweetened drinks is linked to a form of nonalcoholic fatty liver disease in children and adolescents.
Researchers found that children and adolescents who consumed high amounts of fructose, most commonly from sweetened drinks were more likely to have nonalcoholic steatohepatitis (NASH), a form of nonalcoholic fatty liver disease (NAFLD).
Senior investigator Dr. Valerio Nobili, of the Bambino Gesù Hospital in Italy, and colleagues recently reported their findings in the Journal of Hematology.
NAFLD is a condition characterized by a buildup of fat in the liver that is not caused by alcohol consumption. In NASH, the fat buildup is accompanied by inflammation and liver cell damage, which can cause scarring, or fibrosis. NASH can lead to severe complications, including cirrhosis and liver cancer.
Excess weight is a leading risk factor for NAFLD and NASH. According to Dr. Nobili and colleagues, estimates suggest that up to 9.6 percent of all children and 38 percent of obese children in Western countries have some form of liver disease, including NASH.
Assessing the impact of fructose intake on NASH.
According to the researchers, previous studies have indicated that dietary fructose intake leads to an increase in blood levels of uric acid, and high levels of both fructose and uric acid have been identified in people with NAFLD.
For this latest study, Dr. Nobili and team set out to investigate whether fructose and uric acid might be independently associated with NASH.
Numerous studies have shown that high uric acid levels are associated with metabolic syndrome and NAFLD, but to date, to the best of our knowledge

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link: New research provides further evidence of the harms of consuming too many sweetened beverages, after linking soda and other fructose-containing products with increased risk of liver disease.

Researchers suggest that high fructose intake, particularly from sweetened drinks is linked to a form of nonalcoholic fatty liver disease in children and adolescents.
Researchers found that children and adolescents who consumed high amounts of fructose, most commonly from sweetened drinks were more likely to have nonalcoholic steatohepatitis (NASH), a form of nonalcoholic fatty liver disease (NAFLD).
Senior investigator Dr. Valerio Nobili, of the Bambino Gesù Hospital in Italy, and colleagues recently reported their findings in the Journal of Hematology.
NAFLD is a condition characterized by a buildup of fat in the liver that is not caused by alcohol consumption. In NASH, the fat buildup is accompanied by inflammation and liver cell damage, which can cause scarring, or fibrosis. NASH can lead to severe complications, including cirrhosis and liver cancer.
Excess weight is a leading risk factor for NAFLD and NASH. According to Dr. Nobili and colleagues, estimates suggest that up to 9.6 percent of all children and 38 percent of obese children in Western countries have some form of liver disease, including NASH.
Assessing the impact of fructose intake on NASH.
According to the researchers, previous studies have indicated that dietary fructose intake leads to an increase in blood levels of uric acid, and high levels of both fructose and uric acid have been identified in people with NAFLD.
For this latest study, Dr. Nobili and team set out to investigate whether fructose and uric acid might be independently associated with NASH.
Numerous studies have shown that high uric acid levels are associated with metabolic syndrome and NAFLD, but to date, to the best of our knowledg

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